[外语类试卷]专业英语八级(阅读)模拟试卷105及答案与解析.doc
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1、专业英语八级(阅读)模拟试卷 105及答案与解析 SECTION A MULTIPLE-CHOICE QUESTIONS In this section there are several passages followed by fourteen multiple-choice questions. For each multiple-choice question, there are four suggested answers marked A , B, C and D. Choose the one that you think is the best answer. 0 (1)Th
2、at smoking causes lung cancer is well established. But what causes smoking? This is the question at the heart of a study published in Nature by a group of researchers who work at deCODE, an Icelandic genomics company. They do not, quite, answer it. But they do think they have the answer to the relat
3、ed question of why some smokers smoke in moderation whereas others are rarely found without a fag in their hands and thus why some people are, genetically speaking, more susceptible to lung cancer than others. (2)That answer lies in part of human chromosome 15, and depends on what is known as allele
4、 T of SNP rsl051730. A SNP, or single-nucleotide polymorphism, to give its full name(the short version is pronounced “snip“), is a place where genomes routinely differ from one another by a single genetic letter. (3)In this case, the variation happens inside a gene for one of the receptor molecules
5、that nicotine attaches itself to when it produces its buzz. Based on a study of 13,945 Icelandic smokers, deCODEs researchers showed that having a T in the appropriate part of the gene correlates very strongly indeed with being a heavy smoker. The team estimates that the chance of their being wrong
6、is less than one in a thousand trillion. (4)Not surprisingly, having the T variant also correlates with the chance of a smoker getting lung cancer. Each copy(there may be none, one or two, since one can come from a persons father and one from his mother)increases that chance by 30%. The T variant do
7、es not, however, increase the likelihood that someone will take up smoking in the first place. That is either a matter of free will or, if it is genetic, is controlled by genes somewhere else. It all looks neat and simple and extremely plausible. Genes promote smoking; smoking promotes cancer. Howev
8、er, it might be wrong, for another paper in Nature, and a third in its sister journal Nature Genetics, report similar studies that have drawn rather different conclusions. (5)Paul Brennan and Christopher Amos both agree mat something significant is going on in the part of chromosome 15 studied by de
9、CODE. But they have concluded that genetic variation there acts directly on a persons susceptibility to lung cancer, rather than acting indirectly by modifying his smoking behavior. That does not mean the gene or genes in question actually cause lung cancer. Rather, it means that they amplify the ef
10、fects of smoking instead of the amount of smoking. (6)Like deCODE, both Dr. Brennan(who works at the International Agency for Research on Cancer, in France)and Dr. Amos(who works at the University of Texas)identified rsl051730 as an important SNP. Unlike deCODE, though, both think a second SNP, rs80
11、34191, is involved as well. That, and the fact that the region of chromosome 15 under scrutiny has two other nicotine-receptor genes in it, suggests the situation may indeed be more complex. (7)On top of this, Dr. Brennan and Dr. Amos both used a different method from deCODEs. They compared lung-can
12、cer patients directly with otherwise similar cancer-free smokers, in what is known as a case controlled study, and concluded that genetic variants in the nicotine-receptor-rich part of chromosome 15 are changing not smokers behavior, but their susceptibility to cancer. Moreover, Dr. Brennan also cla
13、ims to have discovered an increased susceptibility to lung cancer in non-smokers with the relevant SNPs, though his sample size is small and his result is not supported by Dr. Amoss work. (8)These contradictory conclusions are both puzzling and intriguing. DeCODE has one further piece of evidence in
14、 its favor. Besides the correlation with lung cancer, the T variant also seemed to correlate with peripheral arterial disease, another common side-effect of smoking. On the other hand, the firm also acknowledges that the link it thinks it has discovered does not account for the whole of the risk of
15、smoking-induced lung cancer. What is not in doubt, however, is that there is some sort of a link between genetics and lung cancer. (9)That raises interesting issues, particularly as genetic testing becomes easier. DeCODE has already announced it will add rsl051730 to the standard screen it offers to
16、 those who wish to know their susceptibility to diseases. The day is not far off, therefore, when those who take the essentially irrational decision to start smoking tobacco will be able to find out in advance exactly how foolish they are being. 1 All of the following have enough evidence EXCEPT tha
17、t _. ( A) lung cancer is related to chromosome 15 ( B) lung cancer is related to genetics ( C) smoking is genetically decided ( D) smoking causes lung cancer 2 The results of the study done by Paul Brennan and Christopher Amos _. ( A) have shown a completely contradictory situation with the study do
18、ne by deCODE ( B) suggest that the situation may be more complex than deCODE has identified ( C) show that rs8034191 instead of rs1051730 is related to the susceptibility to lung cancer ( D) smoking behavior is modified by genetic variations in the region of chromosome 15 3 The current conclusions d
19、rawn from the studies have been _. ( A) disappointing ( B) frustrating ( C) enlightening ( D) comprehensive 4 Which of the following is INCORRECT about the study of genetics? ( A) The different methods adopted have led to the different conclusions that researchers drew. ( B) The two groups of resear
20、chers have provided evidence in their favor respectively. ( C) The role of genetics in smoking and lung cancer has yet to be studied and confirmed. ( D) The development of genetic testing has made studies on genetics and lung cancer easier. 4 (1)When outsourcing company Keane Inc. hired Jyoti Taneja
21、 10 months ago to work in its Gurgaon, India, offices, her parents were worried. The 22-year-old Ms. Taneja would be taking calls from American customers of U.S. health-insurance companies, finishing her shift at 2:30 a.m. (2)Hoping to reassure their young hires parents about her safety, her supervi
22、sors at Keane paid several visits to Ms. Tanejas family home in nearby New Delhi. (3)They outlined the security measures Keane takes to protect employees, such as providing a shuttle service to and from work, and having security escorts accompany young women who are riding home alone. “All of that a
23、dds to my mom and dads confidence,“ Ms. Taneja says. (4)Facing a shortage of workers, Indias outsourcing specialists are pulling out the stops. In a culture where twenty-somethings often live with their parents and seek their blessing on major life decisions, family outreach has become a critical re
24、cruiting and retention tool, along with offering more-flexible work hours, higher salaries and continuing education. (5)Such efforts “create a bond, not just between the employees and the organization, but also with the families, and is key to our retention strategy,“ said Ritu Anand, a human-resour
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