大学英语六级分类模拟题452及答案解析.doc
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1、大学英语六级分类模拟题 452 及答案解析(总分:445.00,做题时间:90 分钟)一、Reading Comprehensio(总题数:0,分数:0.00)二、Section A(总题数:1,分数:70.00)Why Teenagers Act CrazyA. Adolescence is practically synonymous in our culture with risk taking, emotional drama and all forms of strange behavior. Until very recently, the widely accepted expl
2、anation for adolescent angst has been psychological. Developmentally, teenagers face a number of social and emotional challenges, like starting to separate from their parents, getting accepted into a peer group and figuring out who they really are. It doesn“t take a psychoanalyst to realize that the
3、se are anxiety-provoking transitions. B. But there is a darker side to adolescence that, until now, was poorly understood: a surge during teenage years in anxiety and fearfulness. Largely because of a quirk (古怪) of brain development, adolescents, on average, experience more anxiety and fear and have
4、 a harder time learning how not to be afraid than either children or adults. C. Different regions and circuits of the brain mature at very different rates. It turns out that the brain circuit for processing fearthe amygdala (杏仁核)is precocious (早熟的) and develops way ahead of the prefrontal cortex (前额
5、皮质), the seat of reasoning and executive control. This means that adolescents have a brain that is wired with an enhanced capacity for fear and anxiety, but is relatively underdeveloped when it comes to calm reasoning. D. You may wonder why, if adolescents have such enhanced capacity for anxiety, th
6、ey are such novelty seekers and risk takers. It would seem that the two traits are at odds. The answer, in part, is that the brain“s reward center, just like its fear circuit, matures earlier than the prefrontal cortex. That reward center drives much of teenagers“ risky behavior. This behavioral par
7、adox also helps explain why adolescents are particularly prone to injury and trauma. The top three killers of teenagers are accidents, homicide and suicide. The brain-development lag has huge implications for how we think about anxiety and how we treat it. It suggests that anxious adolescents may no
8、t be very responsive to psychotherapy that attempts to teach them to be unafraid, like cognitive behavior therapy, which is zealously prescribed for teenagers. E. What we have learned should also make us think twiceand then someabout the ever rising use of stimulants in young people, because these d
9、rugs may worsen anxiety and make it harder for teenagers to do what they are developmentally supposed to do: Learn to be unafraid when it is appropriate to do so. Of course, most adolescents do not develop anxiety disorders, but acquire the skill to modulate (调节) their fear as their prefrontal corte
10、x matures in young adulthood, at around age 25. But up to 20 percent of adolescents in the United States experience a diagnosable anxiety disorder, like generalized anxiety or panic attacks, probably resulting from a mix of genetic factors and environmental influences. The prevalence of anxiety diso
11、rders and risky behavior (both of which reflect this developmental disjunction in the brain) have been relatively steady, which suggests to me that the biological contribution is very significant. F. One of my patients, a 32-year-old man, recalled feeling anxious in social gatherings as a teenager.
12、“It was viscerally (出自内心地) unpleasant and I felt as if I couldn“t even speak the same language as other people in the room,“ he said. It wasn“t that he disliked human company; rather, socializing in groups felt dangerous, even though intellectually he knew that wasn“t the case. He developed a strate
13、gy to deal with his discomfort: alcohol. When he drank, he felt relaxed and able to engage. Now treated and sober for several years, he still has a trace of social anxiety and still wishes for a drink in anticipation of socializing. G. Of course, we all experience anxiety. Among other things, it“s a
14、 normal emotional response to threatening situations. The hallmark of an anxiety disorder is the persistence of anxiety that causes intense distress and interferes with functioning even in safe settings, long after any threat has receded. We“ve recently learned that adolescents show heightened fear
15、responses and have difficulty learning how not to be afraid. In one study using brain M. R. I., researchers at Weill Cornell Medical College and Stanford University found that when adolescents were shown fearful faces, they had exaggerated responses in the amygdala compared with children and adults.
16、 H. The amygdala is a region buried deep beneath the cortex that is critical in evaluating and responding to fear. it sends and receives connections to our prefrontal cortex alerting us to danger even before we have had time to really think about it. Think of that split-second adrenaline (肾上腺素) surg
17、e when you see what appears to be a snake out on a hike in the woods. That instantaneous fear is your amygdala in action. Then you circle back, take another look and this time your prefrontal cortex tells you it was just a harmless stick. Fear learning lies at the heart of anxiety and anxiety disord
18、ers. This primitive form of learning allows us to form associations between events and specific cues and environments that may predict danger. Way back on the savanna (热带草原), for example, we would have learned that the rustle in the grass or the sudden flight of birds might signal a predatorand take
19、n the cue and run to safety. Without the ability to identify such danger signals, we would have been lunch long ago. But once previously threatening cues or situations become safe, we have to be able to re-evaluate them and suppress our learned fear associations. I. Another patient I saw in consulta
20、tion recently, a 23-year-old woman, described how she became anxious when she was younger after seeing a commercial about asthma (哮喘). “It made me incredibly worried for no reason, and I had a panic attack soon after seeing it,“ she said. As an older teenager, she became worried about getting too cl
21、ose to homeless people and would hold her breath when near them, knowing that “this was crazy and made no sense“. B. J. Casey, a professor of psychology and the director of the Sackler Institute at Weill Cornell Medical College, has studied fear learning in a group of children, adolescents and adult
22、s. Subjects were shown a colored square at the same time that they were exposed to an aversive (令人反感的) noise. The colored square, previously a neutral stimulus, became associated with an unpleasant sound and elicited a fear response similar to that elicited by the sound. J. What Dr. Casey and her co
23、lleagues found was that there were no differences between the subjects in the acquisition of fear conditioning. But when Dr. Casey trained the subjects to essentially unlearn the association between the colored square and the noisea process called fear extinctionsomething very different happened. Wi
24、th fear extinction, subjects are repeatedly shown the colored square in the absence of the noise. Now the square, also known as the conditioned stimulus, loses its ability to elicit a fear response. Dr. Casey discovered that adolescents had a much harder time “unlearning“ the link between the colore
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